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goat anti vegfr3 antibody  (R&D Systems)


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    Structured Review

    R&D Systems goat anti vegfr3 antibody
    ( A ) Schematics of the Prox1-CreER T2 and Kras LSL-G12D alleles. ( B ) Schematic showing when mice received tamoxifen (50 μg; p.o.). Tissues were collected when mice were 21 days old. ( C ) Low- and high-magnification views of <t>Vegfr3-positive</t> lymphatic vessels in 100-μm-thick lung sections from control and Kras G12D mice. The high-magnification images are of the boxed regions. ( D ) The diameter of lymphatic vessels was significantly greater in Kras G12D mice (25.65 ± 1.070 μm; n = 5) than in control (Ctrl) mice (14.31 ± 0.9752 μm; n = 5). Data are presented as mean ± SEM. **** P < 0.0001 by 2-tailed, unpaired Student’s t test. Scale bars: 500 μm (low magnification) and 50 μm (high magnification).
    Goat Anti Vegfr3 Antibody, supplied by R&D Systems, used in various techniques. Bioz Stars score: 93/100, based on 15 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/goat+anti+edar/pmc11949019-281-3-6?v=R%26D+Systems
    Average 93 stars, based on 15 article reviews
    goat anti vegfr3 antibody - by Bioz Stars, 2026-08
    93/100 stars

    Images

    1) Product Images from "A single-cell atlas of normal and KRAS G12D -malformed lymphatic vessels"

    Article Title: A single-cell atlas of normal and KRAS G12D -malformed lymphatic vessels

    Journal: JCI Insight

    doi: 10.1172/jci.insight.185181

    ( A ) Schematics of the Prox1-CreER T2 and Kras LSL-G12D alleles. ( B ) Schematic showing when mice received tamoxifen (50 μg; p.o.). Tissues were collected when mice were 21 days old. ( C ) Low- and high-magnification views of Vegfr3-positive lymphatic vessels in 100-μm-thick lung sections from control and Kras G12D mice. The high-magnification images are of the boxed regions. ( D ) The diameter of lymphatic vessels was significantly greater in Kras G12D mice (25.65 ± 1.070 μm; n = 5) than in control (Ctrl) mice (14.31 ± 0.9752 μm; n = 5). Data are presented as mean ± SEM. **** P < 0.0001 by 2-tailed, unpaired Student’s t test. Scale bars: 500 μm (low magnification) and 50 μm (high magnification).
    Figure Legend Snippet: ( A ) Schematics of the Prox1-CreER T2 and Kras LSL-G12D alleles. ( B ) Schematic showing when mice received tamoxifen (50 μg; p.o.). Tissues were collected when mice were 21 days old. ( C ) Low- and high-magnification views of Vegfr3-positive lymphatic vessels in 100-μm-thick lung sections from control and Kras G12D mice. The high-magnification images are of the boxed regions. ( D ) The diameter of lymphatic vessels was significantly greater in Kras G12D mice (25.65 ± 1.070 μm; n = 5) than in control (Ctrl) mice (14.31 ± 0.9752 μm; n = 5). Data are presented as mean ± SEM. **** P < 0.0001 by 2-tailed, unpaired Student’s t test. Scale bars: 500 μm (low magnification) and 50 μm (high magnification).

    Techniques Used: Control



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    ( A ) Schematics of the Prox1-CreER T2 and Kras LSL-G12D alleles. ( B ) Schematic showing when mice received tamoxifen (50 μg; p.o.). Tissues were collected when mice were 21 days old. ( C ) Low- and high-magnification views of <t>Vegfr3-positive</t> lymphatic vessels in 100-μm-thick lung sections from control and Kras G12D mice. The high-magnification images are of the boxed regions. ( D ) The diameter of lymphatic vessels was significantly greater in Kras G12D mice (25.65 ± 1.070 μm; n = 5) than in control (Ctrl) mice (14.31 ± 0.9752 μm; n = 5). Data are presented as mean ± SEM. **** P < 0.0001 by 2-tailed, unpaired Student’s t test. Scale bars: 500 μm (low magnification) and 50 μm (high magnification).
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    ( A ) Schematics of the Prox1-CreER T2 and Kras LSL-G12D alleles. ( B ) Schematic showing when mice received tamoxifen (50 μg; p.o.). Tissues were collected when mice were 21 days old. ( C ) Low- and high-magnification views of <t>Vegfr3-positive</t> lymphatic vessels in 100-μm-thick lung sections from control and Kras G12D mice. The high-magnification images are of the boxed regions. ( D ) The diameter of lymphatic vessels was significantly greater in Kras G12D mice (25.65 ± 1.070 μm; n = 5) than in control (Ctrl) mice (14.31 ± 0.9752 μm; n = 5). Data are presented as mean ± SEM. **** P < 0.0001 by 2-tailed, unpaired Student’s t test. Scale bars: 500 μm (low magnification) and 50 μm (high magnification).
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    Image Search Results


    ( A ) Schematics of the Prox1-CreER T2 and Kras LSL-G12D alleles. ( B ) Schematic showing when mice received tamoxifen (50 μg; p.o.). Tissues were collected when mice were 21 days old. ( C ) Low- and high-magnification views of Vegfr3-positive lymphatic vessels in 100-μm-thick lung sections from control and Kras G12D mice. The high-magnification images are of the boxed regions. ( D ) The diameter of lymphatic vessels was significantly greater in Kras G12D mice (25.65 ± 1.070 μm; n = 5) than in control (Ctrl) mice (14.31 ± 0.9752 μm; n = 5). Data are presented as mean ± SEM. **** P < 0.0001 by 2-tailed, unpaired Student’s t test. Scale bars: 500 μm (low magnification) and 50 μm (high magnification).

    Journal: JCI Insight

    Article Title: A single-cell atlas of normal and KRAS G12D -malformed lymphatic vessels

    doi: 10.1172/jci.insight.185181

    Figure Lengend Snippet: ( A ) Schematics of the Prox1-CreER T2 and Kras LSL-G12D alleles. ( B ) Schematic showing when mice received tamoxifen (50 μg; p.o.). Tissues were collected when mice were 21 days old. ( C ) Low- and high-magnification views of Vegfr3-positive lymphatic vessels in 100-μm-thick lung sections from control and Kras G12D mice. The high-magnification images are of the boxed regions. ( D ) The diameter of lymphatic vessels was significantly greater in Kras G12D mice (25.65 ± 1.070 μm; n = 5) than in control (Ctrl) mice (14.31 ± 0.9752 μm; n = 5). Data are presented as mean ± SEM. **** P < 0.0001 by 2-tailed, unpaired Student’s t test. Scale bars: 500 μm (low magnification) and 50 μm (high magnification).

    Article Snippet: We used a goat anti-Vegfr3 antibody (R&D Systems, AF745; 1:250).

    Techniques: Control